日誌
帕金森氏症、主動脈剝離、唐氏症可能都是細菌感染造成的
所有疾病可能都是細菌附著細胞，干擾基因表達造成的一連串證據。
用治療帕金森氏症的概念來解決手抖，要怎麼做(建議提升IL-10、IL-2、IFN-r)
告訴我們：
為了增加血糖，胰高血糖素(Glucagon)通過增加糖原分解和糖異生來促進肝葡萄糖輸出
葡萄糖刺激的胰島細胞中升高磷脂酸
磷脂酸 (PA)介導的 ATP13A2 激活
ATP13A2功能的喪失導致一般溶酶體缺乏並導致帕金森病神經變性
因此為了增加血糖(葡萄糖)，就必須提升胰高血糖素(Glucagon)
由以上可知
除了IL-2促進胰高血糖素分泌
可能IFN-r也可以促進胰高血糖素分泌
Gut bacteria responsible for increased H2S production, especially the mucus-associated species of the bacterial genera belonging to the Desulfovibrionaceae and Enterobacteriaceae families, are likely play a role in the pathogenesis of Parkinson's disease.
負責增加 H2S 產量的腸道細菌，尤其是屬於脫硫弧菌科和腸桿菌科的細菌屬的粘液相關物種，可能在帕金森病的發病機制中發揮作用。
Desulfovibrio Bacteria Are Associated With Parkinson's Disease
脫硫弧菌與帕金森病有關
Meta-analysis of the Parkinson’s disease gut microbiome suggests alterations linked to intestinal inflammation
帕金森病腸道微生物組的薈萃分析表明與腸道炎症相關的改變
Parkinson's disease may start in the gut and spread to the brain
帕金森病可能從腸道開始並擴散到大腦
主動脈剝離的中醫、西醫論點
Salmonella and S. aureus are regarded as the most common bacterial pathogens associated with mycotic aortic aneurysm.
沙門氏菌和金黃色葡萄球菌被認為是與黴菌性主動脈瘤相關的最常見的細菌病原體。
Clostridium septicum-infected Stanford type A acute aortic dissection: a case report
敗血梭菌感染的Stanford A型急性主動脈剝離一例
Down syndrome (DS)
唐氏綜合症 (DS)
B. forsythus, T. denticola, P. nigrescens, and C. rectus were significantly prevalent throughout all age brackets of the DS children
B. forsythus、T. denticola、P. nigrescens 和 C. rectus 在 DS 兒童的所有年齡段中都非常普遍
The occurrence of P. gingivalis was also significant in the DS subjects over 5 years old. A cluster analysis of the microbial profiles of the DS subjects showed that gingivitis severity was associated with increased varieties of the harboring pathogens and the distribution of P. gingivalis.
牙齦卟啉單胞菌的發生在 5 歲以上的 DS 受試者中也很顯著。 對 DS 受試者微生物特徵的聚類分析表明，牙齦炎的嚴重程度與攜帶病原體的種類增加和牙齦卟啉單胞菌的分佈有關。
By contrast, in the Down syndrome group, both Actinobacteria and Proteobacteria showed high abundance at all three dental stages
相比之下，在唐氏綜合症組中，放線菌和變形菌在所有三個牙科階段都表現出高豐度
